A Rare, Historic Porsche Racer Leads RM Sotheby’s New German Sale
3 min
The 24-year-old actor James Dean died in a car accident, colliding with a college student at a California intersection on the evening of Sept. 30, 1955. The car he was driving was a Porsche, but not an ordinary 356. It was a very streamlined 550 Spyder, nicknamed “Little Bastard” by the race-crazy Dean.
The 550 Spyder was an out-and-out racer, but the kind that owners could register and drive to and from the track in those days. The open-topped Porsche was made for only three years, from 1953 to 1956, and although they were very successful in competition, only 90 were produced. The mid-mounted “Carrera” engine in the 550 had four overhead camshafts and dual ignition. With twin Solex carburetors, it produced 110 horsepower. That wasn’t a lot, but the 550 Spyder was a very light car, just 590 kilograms (1,300 pounds).
An example of the 550 Spyder, from 1955 with colourful racing history, is one of the cars that will be sold by RM Sotheby’s in an auction by Lake Tegernsee, about 40 minutes south of Munich, on July 27. Also on the block is a pair of modern Bugattis, a rare Mercedes-Benz SLR McLaren Stirling Moss, and a 2006 Porsche Carrera GT. The auction is taking place in partnership with the new Concours of Elegance Germany in Bavaria, held July 22-27.
RM Sotheby’s
This Porsche 550 Spyder, with coachwork by Wendler (which also had its hand in the 718 sport racing cars), was delivered to Portugal and competed in European racing circuits. Originally white with burgundy accents, the car was first owned by Fernando Mascarenhas, who achieved class podium positions in races at Barajas and Monsanto in 1955. The 550 then went to Germany that summer for the Nürburgring 500 Kilometres, but the race was cut short because of an accident.
The second owner was Cypriano Flores in 1958. Flores’ son eventually returned the car to Porsche, which did the mechanical work while Wendler restored the body.
Despite the racing, which often results in swapped engines and other components, the 550 still boasts its original chassis, four-cam Carrera motor, and gearbox. The car was restored by Porsche and its original coachbuilder, Wendler, in the early 1990s—and not driven since then. During the restoration, the car’s color was changed to silver, and the interior from beige vinyl to black leather. The pre-auction estimate is €3.5 million to €4.2 million (US$3.78 million to US$4.54 million).
Also to be auctioned at Tegernsee is the aforementioned 2010 Mercedes SLR McLaren Stirling Moss, a virtually unused example with just 45 kilometres on the odometer. First shown in 2009, it was a tribute to the late racing driver’s win in a 300 SLR Mercedes at the 1955 Mille Miglia.
The auction SLR features a lightweight carbon-fibre structure and a supercharged, 5.4-litre V8 with 641 horsepower. A mere 75 Stirling Moss cars were built, and only offered to customers who already owned an SLR McLaren. Without a roof or windshield, the Moss edition was 200 kilograms lighter than the standard car. It could reach 62 miles per hour in 3.5 seconds. The pre-sale estimate is €3.2 million to €3.8 million.
The modern Bugattis include a 2019 Chiron Sport “110 Ans Bugatti” edition, one of 20. The odometer reads only 1,461 kilometres. It’s estimated at €3.3 million to €3.8 million. The other one is the 2010 Veyron 16.4 Grand Sport “Soleil de Nuit,” a one-off Veyron in two-tone black/blue metallic sold new to the royal family of Kuwait. The estimate is €1.5 million to €2 million.
The 2006 Porsche Carrera GT, one of just 1,270 of these race-derived high-performance cars, is also a low-mileage example in silver metallic with 35,698 kilometres showing. It’s powered by a 5.7-litre V10 engine and could reach 62 miles per hour in 3.57 seconds and had a top speed of 205 mph. This one was supplied to Porsche in Leipzig, and a succession of owners barely used it. In 2001, the Porsche benefitted from a major €27,000 service that included a clutch replacement. It’s estimated at €975,000 to €1.275 million.
Porsche collectors might also want to visit the Bonhams|Cars Quail auction during Monterey Car Week starting Aug. 16. The lots include a one-of-62 1971 Porsche 911 S/T (estimated between US$900,000 and US$1.2 million); and a 1993 959 “Komfort” model, one of six, estimated at US$1.5 million to US$2 million.
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At least for people who carry the APOE4 genetic variant, a juicy steak could keep the brain healthy.
3 min
Must even steak be politicised? The American Heart Association recently recommended eating more “plant-based” protein in a move to counter the Health and Human Services Department’s new guidelines calling for more red meat.
Few would argue that eating a Big Mac a day is good for you.
On the other hand, growing evidence, including a study last month in the Journal of the American Medical Association, suggests that eating more meat—particularly unprocessed red meat—can reduce the risk of Alzheimer’s in the quarter or so of people with a particular genetic predisposition.
The APOE4 gene variant is one of the biggest risk factors for Alzheimer’s.
You inherit one copy of the APOE gene from each parent. The most common variant is APOE3; the least is APOE2.
The latter carries a lower risk of Alzheimer’s, while the former is neutral. A quarter of people carry one copy of the APOE4 variant, and about 2% carry two.
APOE4 is more common among people with Northern European and African ancestry. In Europe the variant increases with latitude, and is present in as many as 27% of people in northern countries versus 4% in southern ones. God smiled on the Italians and Greeks.
For unknown reasons, the APOE4 variant increases the risk of Alzheimer’s far more for women than men.
Women’s risk multiplies roughly fourfold if they have one copy and tenfold if they have two. Men with a single copy show little if any higher risk, while those with two face four times the risk.
What makes APOE4 so pernicious? Scientists don’t know exactly, but the variant is also associated with higher cholesterol levels—even among thin people who eat healthily.
Scientists have found that cholesterol builds up in brain cells of APOE4 carriers, which can disrupt communications between neurons and generate amyloid plaque, an Alzheimer’s hallmark.
The Heart Association’s recommendation to eat less red meat may be sound advice for people with high cholesterol caused by indulgent diets.
But a diet high in red meat may be better for the brains of APOE4 carriers.
In the JAMA study, researchers at Sweden’s Karolinska Institute examined how diet, particularly meat consumption, affects dementia risk among seniors with the different APOE variants.
Higher consumption of meat, especially unprocessed red meat, was associated with significantly lower dementia risk for APOE4 carriers.
APOE4 carriers who consumed the most meat—the equivalent of 4.5 ounces a day—were no more likely to develop dementia than noncarriers. (
The study controlled for other variables that are known to affect Alzheimer’s risk including sex, age, physical activity, smoking, alcohol consumption and education.)
APOE4 carriers who ate the most unprocessed meat were at significantly lower risk of dying over the study’s 15-year period and had lower cholesterol than carriers who ate less. Go figure. Noncarriers, however, didn’tenjoy similar benefits from eating more red meat.
The study’s findings are consistent with two large U.K. studies.
One found that each additional 50 grams of red meat (equivalent to half a hamburger patty) that an APOE4 carrier consumed each day was associated with a 36% reduced risk of dementia.
The other found that older women who carried the APOE4 variant and consumed at least one serving a day of unprocessed red meat had a cognitive advantage over carriers who ate less than half a serving, and that this advantage was of roughly equal magnitude to the cognitive disadvantage observed among APOE4 carriers in general.
In all three studies, eating more red meat appeared to negate the increased genetic risk of APOE4.
Perhaps one reason men with the variant are at lower Alzheimer’s risk than women is that men eat more red meat.
These findings might cause chagrin to women who rag their husbands about ordering the rib-eye instead of the heart-healthy salmon.
But remember, the cognitive benefits of eating more red meat appear isolated to APOE4 carriers.
Nutrition is complicated, and categorical recommendations—other than perhaps to avoid nutritionally devoid foods—would best be avoided by governments and health bodies.
Readers can order an at-home test from any number of companies to screen for the APOE4 variant.
The Swedish researchers hypothesize that APOE4 carriers may be evolutionarily adapted to carnivorous diets, since the variant is believed to have emerged between one million and six million years ago during a “hypercarnivorous” period in human history.
The other two APOE variants originated more recently, during eras when humans ate more plants.
APOE4 carriers may absorb more nutrients from meat than plants, the researchers surmise. Vitamin B12—low levels have been associated with cognitive decline—isn’t naturally present in plant-based foods but is abundant in red meat.
Foods high in phytates (such as grains and beans) can interfere with absorption of zinc and iron (also high in red meat), which naturally declines with age. So maybe don’t chuck your steak yet.
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